Prevention Measures of Fatigue in Laying Hens

Caged egg fatigue is also called caged egg hen osteoporosis. It is a systemic skeletal disease in laying hens. Occurs in almost all groups of caged laying flocks, and the prevalence during the laying period ranges from 1% to 10%, which often occurs during the period of high egg production.
First, the cause. Caged chicken fatigue is mainly caused by severe calcium deficiency. High-laying hens are light in weight, low in feed intake, high in feed utilization, and early in sexual maturity. Calcium sources cannot meet the eggshell formation and maintain bone strength, resulting in negative balance of calcium. Overutilized hydrated lime has lower calcium utilization In the hot season, the feed intake of laying hens decreases and the calcium level in feeds does not increase correspondingly; long-term continuous high production requires more calcium, which may lead to cage chicken fatigue. In addition, dietary phosphorus content below the critical value (total phosphorus 0.5%), easily induced in the initial layer of laying chickens, and low calcium and high phosphorus diet may lead to nutritional secondary hyperparathyroidism, so that Depletion of bone calcium.
Second, the symptoms. At the beginning of the disease, soft shell eggs and thin shell eggs were produced, and the breakage rate of eggs increased, but there was no significant change in appetite, spirit and feathers. After that, difficulty in standing, crooked claws, and disordered movements occurred. If timely detection and appropriate treatment measures are taken, most of them can be recovered within 3-5 days. Otherwise, the symptoms will gradually intensify. Eventually, it will result in lameness, failure to stand, sternal sagging, easy rib fracture, and paralysis; The leg bones are fragile.
Third, prevention. 1 Guarantee full-price nutrition and scientific management, so that the broiler chickens reach the optimal weight and body condition when mature. 2 Feed 2%-3% of special pre-opened feed containing calcium in 2-4 weeks before the start of production. When the egg production rate reaches 1%, feed for laying hens should be replaced in time. 3 The content of calcium in the feed of the caged and high-produced layer shall not be lower than 3.5%; and the appropriate ratio of calcium to phosphorus shall be ensured. 4 Provide coarse granules or shell powder to layer hens. The coarse calcium source can account for 1/3-2/3 of total calcium. Calcium source particles larger than 0.75 mm can both increase calcium utilization and avoid calcification in the feed. During the hot season, about 1% of the feed consumption in the afternoon, the coarse-grain calcium is uniformly spread in the feeding trough, which can not only provide enough calcium source, but also stimulate the appetite of the flock and increase the food intake. 5 Normal blood calcium monitoring should be done. Blood calcium should be checked when soft shell eggs are found.
Fourth, treatment. The diseased chickens were singled out, fed individually, supplemented with bone pellets or coarse-grained calcium carbonate and generally cured within 3-5 days. Some diseased chickens that are discontinued are generally recovered in less than one week when they are fed alone and they are guaranteed to eat water. Add 2%-3% coarse calcium carbonate to the feed of the same group of chickens (excluding normal calcium levels), and add 2000 international units of vitamin D3 per kg of diet. After about 2-3 weeks, the blood calcium of the chicken can rise to At normal levels, the incidence is significantly reduced. The calcium-depleted hen's leg bone was fully recalcified after 3 weeks. The supplement of coarse-grain calcium carbonate and vitamin D3 needs to last for about one month. If the condition is found late, it usually takes about 20 days to recover. Individuals with severe disease can die.

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